{"id":283967,"date":"2026-07-29T11:11:11","date_gmt":"2026-07-29T11:11:11","guid":{"rendered":"https:\/\/pharmeasy.in\/blog\/?p=283967"},"modified":"2026-07-29T11:11:20","modified_gmt":"2026-07-29T11:11:20","slug":"diagnostic-g6pd-deficiency-symptoms-causes-diagnosis-treatment-prevention","status":"publish","type":"post","link":"https:\/\/pharmeasy.in\/blog\/diagnostic-g6pd-deficiency-symptoms-causes-diagnosis-treatment-prevention\/","title":{"rendered":"G6PD Deficiency: Symptoms, Causes, Diagnosis, Treatment &amp; Prevention"},"content":{"rendered":"<!DOCTYPE html PUBLIC \"-\/\/W3C\/\/DTD HTML 4.0 Transitional\/\/EN\" \"http:\/\/www.w3.org\/TR\/REC-html40\/loose.dtd\">\n<html><head><meta http-equiv=\"Content-Type\" content=\"text\/html; charset=utf-8\">\n<\/head><body><div id=\"ez-toc-container\" class=\"ez-toc-v2_0_79_2 counter-hierarchy ez-toc-counter ez-toc-grey ez-toc-container-direction\">\n<p class=\"ez-toc-title\" style=\"cursor:inherit\">Table of Contents<\/p>\n<label for=\"ez-toc-cssicon-toggle-item-6a6bcd367abb8\" class=\"ez-toc-cssicon-toggle-label\"><span class=\"ez-toc-cssicon\"><span class=\"eztoc-hide\" style=\"display:none;\">Toggle<\/span><span class=\"ez-toc-icon-toggle-span\"><svg style=\"fill: #999;color:#999\" xmlns=\"http:\/\/www.w3.org\/2000\/svg\" class=\"list-377408\" width=\"20px\" height=\"20px\" viewBox=\"0 0 24 24\" fill=\"none\"><path d=\"M6 6H4v2h2V6zm14 0H8v2h12V6zM4 11h2v2H4v-2zm16 0H8v2h12v-2zM4 16h2v2H4v-2zm16 0H8v2h12v-2z\" fill=\"currentColor\"><\/path><\/svg><svg style=\"fill: #999;color:#999\" class=\"arrow-unsorted-368013\" xmlns=\"http:\/\/www.w3.org\/2000\/svg\" width=\"10px\" height=\"10px\" viewBox=\"0 0 24 24\" version=\"1.2\" baseProfile=\"tiny\"><path d=\"M18.2 9.3l-6.2-6.3-6.2 6.3c-.2.2-.3.4-.3.7s.1.5.3.7c.2.2.4.3.7.3h11c.3 0 .5-.1.7-.3.2-.2.3-.5.3-.7s-.1-.5-.3-.7zM5.8 14.7l6.2 6.3 6.2-6.3c.2-.2.3-.5.3-.7s-.1-.5-.3-.7c-.2-.2-.4-.3-.7-.3h-11c-.3 0-.5.1-.7.3-.2.2-.3.5-.3.7s.1.5.3.7z\"\/><\/svg><\/span><\/span><\/label><input type=\"checkbox\"  id=\"ez-toc-cssicon-toggle-item-6a6bcd367abb8\"  aria-label=\"Toggle\" \/><nav><ul class='ez-toc-list ez-toc-list-level-1 ' ><li class='ez-toc-page-1 ez-toc-heading-level-2'><a class=\"ez-toc-link ez-toc-heading-1\" href=\"https:\/\/pharmeasy.in\/blog\/diagnostic-g6pd-deficiency-symptoms-causes-diagnosis-treatment-prevention\/#Introduction\" >Introduction<\/a><\/li><li class='ez-toc-page-1 ez-toc-heading-level-2'><a class=\"ez-toc-link ez-toc-heading-2\" href=\"https:\/\/pharmeasy.in\/blog\/diagnostic-g6pd-deficiency-symptoms-causes-diagnosis-treatment-prevention\/#What_Is_G6PD_Deficiency\" >What\u00a0Is\u00a0G6PD Deficiency?<\/a><\/li><li class='ez-toc-page-1 ez-toc-heading-level-2'><a class=\"ez-toc-link ez-toc-heading-3\" href=\"https:\/\/pharmeasy.in\/blog\/diagnostic-g6pd-deficiency-symptoms-causes-diagnosis-treatment-prevention\/#Why_Is_G6PD_Important\" >Why Is G6PD Important?<\/a><\/li><li class='ez-toc-page-1 ez-toc-heading-level-2'><a class=\"ez-toc-link ez-toc-heading-4\" href=\"https:\/\/pharmeasy.in\/blog\/diagnostic-g6pd-deficiency-symptoms-causes-diagnosis-treatment-prevention\/#Causes_of_G6PD_Deficiency\" >Causes of G6PD Deficiency<\/a><\/li><li class='ez-toc-page-1 ez-toc-heading-level-2'><a class=\"ez-toc-link ez-toc-heading-5\" href=\"https:\/\/pharmeasy.in\/blog\/diagnostic-g6pd-deficiency-symptoms-causes-diagnosis-treatment-prevention\/#Symptoms_of_G6PD_Deficiency\" >Symptoms of G6PD Deficiency<\/a><\/li><li class='ez-toc-page-1 ez-toc-heading-level-2'><a class=\"ez-toc-link ez-toc-heading-6\" href=\"https:\/\/pharmeasy.in\/blog\/diagnostic-g6pd-deficiency-symptoms-causes-diagnosis-treatment-prevention\/#How_Is_G6PD_Deficiency_Diagnosed\" >How Is G6PD Deficiency Diagnosed?<\/a><\/li><li class='ez-toc-page-1 ez-toc-heading-level-2'><a class=\"ez-toc-link ez-toc-heading-7\" href=\"https:\/\/pharmeasy.in\/blog\/diagnostic-g6pd-deficiency-symptoms-causes-diagnosis-treatment-prevention\/#Treatment_of_G6PD_Deficiency\" >Treatment of G6PD Deficiency<\/a><\/li><li class='ez-toc-page-1 ez-toc-heading-level-2'><a class=\"ez-toc-link ez-toc-heading-8\" href=\"https:\/\/pharmeasy.in\/blog\/diagnostic-g6pd-deficiency-symptoms-causes-diagnosis-treatment-prevention\/#Possible_Complications_of_G6PD_Deficiency\" >Possible Complications of G6PD Deficiency<\/a><\/li><li class='ez-toc-page-1 ez-toc-heading-level-2'><a class=\"ez-toc-link ez-toc-heading-9\" href=\"https:\/\/pharmeasy.in\/blog\/diagnostic-g6pd-deficiency-symptoms-causes-diagnosis-treatment-prevention\/#Prevention_of_Haemolytic_Episodes_in_G6PD_Deficiency\" >Prevention of\u00a0Haemolytic Episodes in\u00a0G6PD Deficiency<\/a><\/li><li class='ez-toc-page-1 ez-toc-heading-level-2'><a class=\"ez-toc-link ez-toc-heading-10\" href=\"https:\/\/pharmeasy.in\/blog\/diagnostic-g6pd-deficiency-symptoms-causes-diagnosis-treatment-prevention\/#When_Should_You_Consult_a_Doctor\" >When Should You Consult a\u00a0Doctor?<\/a><\/li><li class='ez-toc-page-1 ez-toc-heading-level-2'><a class=\"ez-toc-link ez-toc-heading-11\" href=\"https:\/\/pharmeasy.in\/blog\/diagnostic-g6pd-deficiency-symptoms-causes-diagnosis-treatment-prevention\/#Conclusion\" >Conclusion<\/a><\/li><li class='ez-toc-page-1 ez-toc-heading-level-2'><a class=\"ez-toc-link ez-toc-heading-12\" href=\"https:\/\/pharmeasy.in\/blog\/diagnostic-g6pd-deficiency-symptoms-causes-diagnosis-treatment-prevention\/#Frequently_Asked_Questions_FAQs\" >Frequently Asked Questions (FAQs)<\/a><\/li><li class='ez-toc-page-1 ez-toc-heading-level-2'><a class=\"ez-toc-link ez-toc-heading-13\" href=\"https:\/\/pharmeasy.in\/blog\/diagnostic-g6pd-deficiency-symptoms-causes-diagnosis-treatment-prevention\/#References\" >References<\/a><\/li><\/ul><\/nav><\/div>\n    <a rel=\"nofollow\" href=\"https:\/\/pharmeasy.in\/blog\/channel-health-talk\"\n       class=\"pe-healthtalk-banner\"\n       style=\"position:relative; display:block; margin:10px; border-radius:10px; border-bottom:2px solid #117B77; overflow:hidden;\">\n        <span aria-hidden=\"true\" style=\"position:absolute; left:0; bottom:0; width:2px; height:50%; background:#10847e;\"><\/span>\n        <img alt=\"Join Health Talk by PharmEasy on WhatsApp\"\n             src=\"https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/01\/PEhealthtalk-1.png\"\n             style=\"width:100%; display:block; border-radius:10px;\">\n    <\/a>\n<h2 class=\"wp-block-heading\"><span class=\"ez-toc-section\" id=\"Introduction\"><\/span><strong>Introduction<\/strong><span class=\"ez-toc-section-end\"><\/span><\/h2>\n\n\n\n<p>Some inherited conditions can remain unnoticed for years, even when they have been present since birth.\u00a0Glucose-6-phosphate dehydrogenase (G6PD) deficiency is among the most common inherited enzyme disorders worldwide. Many people discover it only after certain medicines, foods, or infections trigger symptoms<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a>,<a href=\"https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC4571844\/\" target=\"_blank\" rel=\"noreferrer noopener\">2<\/a><\/sup>.\u00a0<\/p>\n\n\n\n<p>Most people\u00a0with G6PD deficiency live healthy, normal lives,\u00a0but\u00a0recognising triggers is\u00a0important for preventing\u00a0complications.\u00a0This\u00a0blog\u00a0covers\u00a0the symptoms, causes, diagnosis, treatment, and practical tips\u00a0for understanding\u00a0and\u00a0managing\u00a0G6PD deficiency.\u00a0<\/p>\n\n\n\n<h2 class=\"wp-block-heading\"><span class=\"ez-toc-section\" id=\"What_Is_G6PD_Deficiency\"><\/span><strong>What\u00a0Is\u00a0G6PD Deficiency?<\/strong><span class=\"ez-toc-section-end\"><\/span><\/h2>\n\n\n\n<p>G6PD deficiency is an inherited genetic condition in which the body does not produce enough of the G6PD enzyme, or the\u00a0enzyme has reduced\u00a0activity<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a><\/sup>.\u00a0When people with G6PD deficiency are exposed to certain triggers\u00a0(such as specific medicines, infections, or foods like fava beans),\u00a0their red blood cells\u00a0(RBCs)\u00a0can break down faster than the body can replace them. This process\u00a0(called haemolysis)\u00a0can lead to haemolytic\u00a0anaemia (a condition in which the body does not have enough healthy\u00a0RBCs\u00a0to carry oxygen effectively)<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a><\/sup>.<\/p>\n\n\n\n<div class=\"wp-block-group fact-box\"><div class=\"wp-block-group__inner-container is-layout-constrained wp-block-group-is-layout-constrained\">\n<p>Did You Know?<\/p>\n\n\n\n<ul>\n<li>It is one of the most common inherited enzyme deficiencies worldwide, affecting an estimated 400 million people globally<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a>,<a href=\"https:\/\/medlineplus.gov\/genetics\/condition\/glucose-6-phosphate-dehydrogenase-deficiency\/\" target=\"_blank\" rel=\"noreferrer noopener\">3<\/a><\/sup>.\u00a0<\/li>\n\n\n\n<li>Around 5% of the world\u2019s population carries a G6PD gene variant, although not everyone develops symptoms. Because the G6PD gene is\u00a0located\u00a0on the X chromosome, the condition is more common in males, while females may carry the altered gene with or without developing symptoms<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a>,<a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/34551338\/\" target=\"_blank\" rel=\"noreferrer noopener\">4<\/a><\/sup>.\u00a0<\/li>\n\n\n\n<li>G6PD deficiency is more common in parts of Africa, the Middle East, the Mediterranean, and Asia.\u00a0It is\u00a0believed\u00a0that\u00a0the mutated gene, which results in unstable RBCs,\u00a0impairs the\u00a0malaria\u00a0parasite\u2019s ability to survive and\u00a0multiply<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a><a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/33543710\/\">,<\/a><a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/33543710\/\" target=\"_blank\" rel=\"noreferrer noopener\">5<\/a><\/sup>.\u00a0<\/li>\n<\/ul>\n<\/div><\/div>\n\n\n\n<h2 class=\"wp-block-heading\"><span class=\"ez-toc-section\" id=\"Why_Is_G6PD_Important\"><\/span><strong>Why Is G6PD Important?<\/strong><span class=\"ez-toc-section-end\"><\/span><\/h2>\n\n\n\n<p>G6PD helps the body produce a molecule called NADPH\u00a0(nicotinamide adenine dinucleotide phosphate)\u00a0through the pentose phosphate pathway. NADPH keeps glutathione, the main antioxidant in\u00a0RBCs, in its active form. This antioxidant protects\u00a0RBCs from\u00a0oxidative stress\u00a0damage and prevents them from breaking down too early<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a>,<a href=\"https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC6683872\/\" target=\"_blank\" rel=\"noreferrer noopener\">6<\/a><\/sup>.\u00a0<\/p>\n\n\n\n<p>Unlike most other cells, mature\u00a0RBCs cannot make new proteins or generate\u00a0additional\u00a0G6PD because they lack a nucleus and other cell structures. As a result, they rely entirely on the G6PD enzyme already present to defend against oxidative\u00a0stress<sup><a href=\"https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC6683872\/\" target=\"_blank\" rel=\"noreferrer noopener\">7<\/a><\/sup>. \u00a0When G6PD activity is too low, exposure to substances that cause oxidative stress, such as certain medications, infections, chemicals, or foods, can damage\u00a0RBCs and cause them to break down prematurely (haemolysis). This may lead to haemolytic anaemia, reducing the\u00a0blood\u2019s\u00a0ability to carry oxygen throughout the body<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a><\/sup>.<\/p><div id=\"sfa_container_283967\" class=\"sfa_container\" style=\"display:none\"><div class=\"sfa_overlay\"><\/div><button id=\"sfa_btn_283967\" class=\"sfa_btn\">Show Full Article<\/button><\/div>\n\n\n\n<h2 class=\"wp-block-heading\"><span class=\"ez-toc-section\" id=\"Causes_of_G6PD_Deficiency\"><\/span><strong>Causes of G6PD Deficiency<\/strong><span class=\"ez-toc-section-end\"><\/span><\/h2>\n\n\n\n<p>G6PD deficiency is caused by inherited changes (mutations) in the G6PD gene, which provides instructions for making the G6PD enzyme. G6PD deficiency is caused by mutations in the G6PD gene, resulting in reduced or absent G6PD enzyme activity<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a><\/sup>.<\/p>\n\n\n\n<p>This mutation is triggered by certain factors such as:\u00a0<\/p>\n\n\n\n<ul>\n<li>Medicines, infections, fava beans, and certain chemicals do not cause G6PD deficiency. Instead, they can trigger symptoms or episodes of haemolysis in people who are already born with the condition<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a><\/sup>.<\/li>\n<\/ul>\n\n\n\n<p><em><strong>Note:<\/strong>\u00a0More than 400 genetic variants of G6PD deficiency have been\u00a0identified. These variants differ\u00a0in the amount of\u00a0enzyme activity they produce, which is why some individuals never develop symptoms, while others experience severe episodes of haemolysis<sup><a href=\"https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC6683872\/\" target=\"_blank\" rel=\"noreferrer noopener\">8<\/a><\/sup>.\u00a0<\/em><\/p>\n\n\n\n<h2 class=\"wp-block-heading\"><span class=\"ez-toc-section\" id=\"Symptoms_of_G6PD_Deficiency\"><\/span><strong>Symptoms of G6PD Deficiency<\/strong><span class=\"ez-toc-section-end\"><\/span><\/h2>\n\n\n\n<p>Most people with G6PD deficiency do not have symptoms unless they are exposed to a trigger. When\u00a0RBCs\u00a0undergo\u00a0haemolysis, symptoms can develop suddenly and range from mild to severe. Common\u00a0G6PD deficiency symptoms\u00a0include<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a>,<a href=\"https:\/\/www.healthdirect.gov.au\/G6PD-deficiency\" target=\"_blank\" rel=\"noreferrer noopener\">9<\/a><\/sup>:\u00a0<\/p>\n\n\n\n<ul>\n<li>Pale skin (pallor)\u00a0<\/li>\n\n\n\n<li>Jaundice (yellowing of the skin and eyes)\u00a0<\/li>\n\n\n\n<li>Dark-coloured urine\u00a0<\/li>\n\n\n\n<li>Fatigue or unusual tiredness\u00a0<\/li>\n\n\n\n<li>Shortness of breath\u00a0<\/li>\n\n\n\n<li>Rapid heartbeat (tachycardia)\u00a0<\/li>\n\n\n\n<li>Abdominal or back pain\u00a0<\/li>\n\n\n\n<li>Fever (particularly if there is an infection)\u00a0<\/li>\n<\/ul>\n\n\n\n<p>Some babies with G6PD deficiency may develop symptoms shortly after birth,\u00a0including<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a><\/sup>:\u00a0<\/p>\n\n\n\n<ul>\n<li>Severe neonatal jaundice that appears within the first few days of life\u00a0<\/li>\n\n\n\n<li>Poor feeding or difficulty feeding\u00a0<\/li>\n\n\n\n<li>Increased or reduced muscle tone\u00a0<\/li>\n\n\n\n<li>Excessive lethargy\u00a0<\/li>\n\n\n\n<li>Seizures\u00a0<\/li>\n<\/ul>\n\n\n\n<p>Rarely, untreated severe jaundice\u00a0in babies\u00a0can lead to kernicterus,\u00a0characterised by permanent\u00a0brain damage<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a><\/sup>.<\/p>\n\n\n\n<h2 class=\"wp-block-heading\"><span class=\"ez-toc-section\" id=\"How_Is_G6PD_Deficiency_Diagnosed\"><\/span><strong>How Is G6PD Deficiency Diagnosed?<\/strong><span class=\"ez-toc-section-end\"><\/span><\/h2>\n\n\n\n<p>Diagnosis typically involves blood tests that measure G6PD enzyme activity. Based on your symptoms, the doctor may rely\u00a0on<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a>,<a href=\"https:\/\/medlineplus.gov\/lab-tests\/g6pd-test\/\" target=\"_blank\" rel=\"noreferrer noopener\">10<\/a><\/sup>:\u00a0<\/p>\n\n\n\n<ul>\n<li>Medical history and physical examination to assess symptoms, family history, recent infections, medications, and exposure to triggers such as fava beans.\u00a0<\/li>\n\n\n\n<li>Blood tests, including a <a href=\"https:\/\/pharmeasy.in\/diagnostics\/profile\/complete-blood-count-cbc-408\" target=\"_blank\" rel=\"noreferrer noopener\"><strong>complete blood coun<\/strong>t<\/a>, <a href=\"https:\/\/pharmeasy.in\/diagnostics\/tests\/bilirubindirect-263\" target=\"_blank\" rel=\"noreferrer noopener\"><strong>bilirubin levels<\/strong>,<\/a> <a href=\"https:\/\/pharmeasy.in\/diagnostics\/tests\/retic-count-124\" target=\"_blank\" rel=\"noreferrer noopener\"><strong>reticulocyte count<\/strong><\/a>, and other tests to detect\u00a0RBC\u00a0haemolysis.\u00a0<\/li>\n\n\n\n<li>A\u00a0<strong><a href=\"https:\/\/pharmeasy.in\/diagnostics\/tests\/g6pd-81\" target=\"_blank\" rel=\"noreferrer noopener\">G6PD test<\/a>\u00a0<\/strong>measures\u00a0how well\u00a0the G6PD enzyme in\u00a0RBCs\u00a0is\u00a0functioning.\u00a0<\/li>\n\n\n\n<li>Genetic testing may be recommended in selected cases to\u00a0identify\u00a0the specific G6PD gene variant.\u00a0<\/li>\n<\/ul>\n\n\n\n<p><em><strong>Note:<\/strong>\u00a0G6PD test results may appear\u00a0normal during or soon after a\u00a0haemolytic\u00a0episode\u00a0because the\u00a0tested sample could contain more of younger RBCs with higher G6PD activity and a lower proportion of\u00a0older\u00a0RBCs with low G6PD levels<sup><a href=\"https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC3678266\/\" target=\"_blank\" rel=\"noreferrer noopener\">11<\/a><\/sup>.\u00a0If G6PD deficiency is still suspected despite a normal test result, your doctor may recommend\u00a0repeating the test after recovery, usually about 3 months later, for a more accurate diagnosis.\u00a0<\/em><\/p>\n\n\n\n<p><strong>Important:<\/strong>\u00a0Babies with unexplained or severe jaundice may be tested for G6PD deficiency, especially if they have a family history or belong to a high-risk population<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a>,<a href=\"https:\/\/www.healthdirect.gov.au\/G6PD-deficiency\" target=\"_blank\" rel=\"noreferrer noopener\">9<\/a><\/sup>.<\/p>\n\n\n\n<h2 class=\"wp-block-heading\"><span class=\"ez-toc-section\" id=\"Treatment_of_G6PD_Deficiency\"><\/span><strong>Treatment of G6PD Deficiency<\/strong><span class=\"ez-toc-section-end\"><\/span><\/h2>\n\n\n<div class=\"wp-block-image\">\n<figure class=\"aligncenter size-large\"><img loading=\"lazy\" decoding=\"async\" width=\"1024\" height=\"572\" src=\"https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Treatment-of-G6PD-Deficiency-1024x572.webp\" alt=\"Treatment of G6PD Deficiency\" class=\"wp-image-284039\" srcset=\"https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Treatment-of-G6PD-Deficiency-1024x572.webp 1024w, https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Treatment-of-G6PD-Deficiency-375x209.webp 375w, https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Treatment-of-G6PD-Deficiency-768x429.webp 768w, https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Treatment-of-G6PD-Deficiency-480x268.webp 480w, https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Treatment-of-G6PD-Deficiency-860x480.webp 860w, https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Treatment-of-G6PD-Deficiency-150x84.webp 150w, https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Treatment-of-G6PD-Deficiency.webp 1376w\" sizes=\"(max-width: 1024px) 100vw, 1024px\" \/><\/figure><\/div>\n\n\n<p>There is no cure for G6PD deficiency. Management\u00a0mainly focuses\u00a0on avoiding triggers and treating haemolysis if it occurs.\u00a0Management\u00a0options\u00a0for\u00a0G6PD deficiency<strong>\u00a0<\/strong>include<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a>,<a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/34389161\/\" target=\"_blank\" rel=\"noreferrer noopener\">12<\/a><\/sup>.\u00a0<\/p>\n\n\n\n<ul>\n<li><strong>Avoiding\u00a0the\u00a0Trigger:<\/strong>\u00a0The first step is to stop or avoid the medicine, food (such as fava beans), or chemical that triggered the haemolytic episode.\u00a0<\/li>\n\n\n\n<li><strong>Managing\u00a0Underlying\u00a0Infections:<\/strong>\u00a0Since infections can cause more oxidative stress, they should be treated promptly.\u00a0<\/li>\n\n\n\n<li><strong>Seeking Timely Blood\u00a0Transfusion:\u00a0<\/strong>People with severe anaemia,\u00a0based on symptoms and haemoglobin level,\u00a0may require a blood transfusion to replace damaged\u00a0RBCs.\u00a0<\/li>\n\n\n\n<li><strong>Phototherapy (for\u00a0Newborns):<\/strong>\u00a0Babies with jaundice may need phototherapy\u00a0thathow to detect g6pd deficiency uses\u00a0specialised blue light\u00a0to break\u00a0down excess bilirubin\u00a0in the baby\u2019s skin into harmless, water-soluble byproducts. Severe cases may require an exchange transfusion to avoid complications.\u00a0<\/li>\n\n\n\n<li><strong>Supportive\u00a0Care:<\/strong>\u00a0Mild cases usually improve\u00a0after\u00a0removing the triggering factor,\u00a0maintaining\u00a0adequate hydration, and close monitoring.\u00a0<\/li>\n\n\n\n<li><strong>Regular follow-up and education:<\/strong>\u00a0People with G6PD deficiency should\u00a0identify\u00a0which\u00a0triggers to avoid and\u00a0should\u00a0inform doctors of their condition before starting any new medication.\u00a0<\/li>\n<\/ul>\n\n\n\n<h2 class=\"wp-block-heading\"><span class=\"ez-toc-section\" id=\"Possible_Complications_of_G6PD_Deficiency\"><\/span><strong>Possible Complications of G6PD Deficiency<\/strong><span class=\"ez-toc-section-end\"><\/span><\/h2>\n\n\n\n<p>Most people with G6PD deficiency remain healthy if they avoid known triggers. However,\u00a0repeated\u00a0or severe episodes of\u00a0RBC\u00a0breakdown can lead to complications,\u00a0including<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a>,<a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/34551338\/\" target=\"_blank\" rel=\"noreferrer noopener\">4<\/a>,<a href=\"https:\/\/www.healthdirect.gov.au\/G6PD-deficiency\" target=\"_blank\" rel=\"noreferrer noopener\">9<\/a><\/sup>:<\/p>\n\n\n\n<ul>\n<li><strong>Acute\u00a0Haemolytic\u00a0Anaemia:<\/strong>\u00a0The most common complication, caused by rapid destruction of\u00a0RBCs after exposure to\u00a0a\u00a0trigger.\u00a0<\/li>\n\n\n\n<li><strong>Severe\u00a0Jaundice in\u00a0Newborns:<\/strong>\u00a0High bilirubin levels can occur in affected newborns. If left unaddressed, severe jaundice may lead to kernicterus, a rare but serious type of brain damage.\u00a0<\/li>\n\n\n\n<li><strong>Favism:<\/strong>\u00a0A severe form of acute haemolytic anaemia that develops after eating fava (broad) beans.\u00a0Although favism can affect people of any age, it is more\u00a0common\u00a0in children,\u00a0particularly boys,\u00a0living in areas where G6PD deficiency is more prevalent.\u00a0<\/li>\n\n\n\n<li><strong>Haemoglobinuria:\u00a0<\/strong>In severe cases,\u00a0RBC\u00a0breakdown may release haemoglobin into the urine, causing it to appear dark or reddish-brown. However, not all haemolytic episodes in G6PD deficiency result in haemoglobinuria.\u00a0<\/li>\n\n\n\n<li><strong>Acute\u00a0Kidney\u00a0Injury:<\/strong>\u00a0In rare cases, severe haemolysis can damage the kidneys and require hospitalisation.\u00a0<\/li>\n\n\n\n<li><strong>Chronic\u00a0Nonspherocytic\u00a0Haemolytic\u00a0Anaemia:\u00a0<\/strong>A small number of people with severe G6PD variants may have ongoing destruction of\u00a0RBCs, even without obvious triggers.\u00a0<\/li>\n<\/ul>\n\n\n\n<h2 class=\"wp-block-heading\"><span class=\"ez-toc-section\" id=\"Prevention_of_Haemolytic_Episodes_in_G6PD_Deficiency\"><\/span><strong>Prevention of\u00a0Haemolytic Episodes in\u00a0G6PD Deficiency<\/strong><span class=\"ez-toc-section-end\"><\/span><\/h2>\n\n\n<div class=\"wp-block-image\">\n<figure class=\"aligncenter size-large\"><img loading=\"lazy\" decoding=\"async\" width=\"1024\" height=\"572\" src=\"https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Prevention-of-Haemolytic-Episodes-in-G6PD-Deficiency-1024x572.webp\" alt=\"Prevention of\u00a0Haemolytic Episodes in\u00a0G6PD Deficiency\" class=\"wp-image-284040\" srcset=\"https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Prevention-of-Haemolytic-Episodes-in-G6PD-Deficiency-1024x572.webp 1024w, https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Prevention-of-Haemolytic-Episodes-in-G6PD-Deficiency-375x209.webp 375w, https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Prevention-of-Haemolytic-Episodes-in-G6PD-Deficiency-768x429.webp 768w, https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Prevention-of-Haemolytic-Episodes-in-G6PD-Deficiency-480x268.webp 480w, https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Prevention-of-Haemolytic-Episodes-in-G6PD-Deficiency-860x480.webp 860w, https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Prevention-of-Haemolytic-Episodes-in-G6PD-Deficiency-150x84.webp 150w, https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Prevention-of-Haemolytic-Episodes-in-G6PD-Deficiency.webp 1376w\" sizes=\"(max-width: 1024px) 100vw, 1024px\" \/><\/figure><\/div>\n\n\n<p>G6PD deficiency itself cannot be prevented because it is an inherited genetic condition. However, episodes of haemolysis can often be prevented by avoiding known triggers<sup><a href=\"https:\/\/www.healthdirect.gov.au\/G6PD-deficiency\" target=\"_blank\" rel=\"noreferrer noopener\">9<\/a><\/sup>:<\/p>\n\n\n\n<ul>\n<li>Avoid trigger medications that can cause\u00a0RBC\u00a0breakdown. Always inform your doctor or pharmacist that you have G6PD deficiency before starting any new medicine.\u00a0<\/li>\n\n\n\n<li>Avoid fava (broad) beans and other foods known to trigger haemolysis.\u00a0<\/li>\n\n\n\n<li>Address infections promptly, as infections are a common trigger for haemolytic episodes.\u00a0<\/li>\n\n\n\n<li>Stay up to date with recommended vaccinations to help reduce the risk of infections.\u00a0<\/li>\n\n\n\n<li>Be cautious with herbal and alternative medicines, as some may increase the risk of haemolysis.\u00a0<\/li>\n\n\n\n<li>Avoid exposure to naphthalene (mothballs) and other oxidising chemicals.\u00a0<\/li>\n\n\n\n<li>Know the warning signs of haemolysis, such as jaundice, dark urine, fatigue, and shortness of breath, and seek medical care promptly if they occur.\u00a0<\/li>\n<\/ul>\n\n\n\n<p><strong><em>Also Read: <a href=\"https:\/\/pharmeasy.in\/blog\/diagnostic-high-creatinine-symptoms-causes-diagnosis-treatment\/\" target=\"_blank\" rel=\"noreferrer noopener\">High Creatinine: Symptoms, Causes, Diagnosis &amp; Treatment<\/a><\/em><\/strong><\/p>\n\n\n\n<h2 class=\"wp-block-heading\"><span class=\"ez-toc-section\" id=\"When_Should_You_Consult_a_Doctor\"><\/span><strong>When Should You Consult a\u00a0Doctor?<\/strong><span class=\"ez-toc-section-end\"><\/span><\/h2>\n\n\n\n<p><a href=\"https:\/\/pharmeasy.in\/online-doctor-consultation\/\" target=\"_blank\" rel=\"noreferrer noopener\"><strong>Seek medical attention\u00a0immediately<\/strong><\/a>\u00a0if you\u00a0(or your child)\u00a0have\u00a0G6PD deficiency and develop signs of haemolysis, especially after exposure to a known trigger. You should see a doctor if you experience<sup><a href=\"https:\/\/www.healthdirect.gov.au\/G6PD-deficiency\" target=\"_blank\" rel=\"noreferrer noopener\">9<\/a><\/sup>:\u00a0<\/p>\n\n\n\n<ul>\n<li>Yellowing of the skin or eyes (jaundice)\u00a0<\/li>\n\n\n\n<li>Dark-coloured urine\u00a0<\/li>\n\n\n\n<li>Pale skin\u00a0<\/li>\n\n\n\n<li>Extreme tiredness, weakness, or lethargy\u00a0<\/li>\n\n\n\n<li>Fever, particularly if accompanied by other symptoms\u00a0<\/li>\n\n\n\n<li>Shortness of breath or rapid heartbeat\u00a0<\/li>\n\n\n\n<li>Dizziness or fainting\u00a0<\/li>\n<\/ul>\n\n\n\n<p><em><strong>Note:<\/strong>\u00a0For newborns,\u00a0seek urgent medical care if your baby develops jaundice within the first 24 hours of life, has difficulty feeding, or appears unusually sleepy. Jaundice that develops after the first 24 hours should also be evaluated by a healthcare provider, as early diagnosis and treatment can help prevent complications<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a>,<a href=\"https:\/\/www.healthdirect.gov.au\/G6PD-deficiency\" target=\"_blank\" rel=\"noreferrer noopener\">9<\/a>,<a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK532930\/\" target=\"_blank\" rel=\"noreferrer noopener\">13<\/a><\/sup><\/em>.<em>\u00a0<\/em><\/p>\n\n\n\n<h2 class=\"wp-block-heading\"><span class=\"ez-toc-section\" id=\"Conclusion\"><\/span><strong>Conclusion<\/strong><span class=\"ez-toc-section-end\"><\/span><\/h2>\n\n\n\n<p>G6PD deficiency is a common inherited condition that often remains unnoticed until exposure to certain triggers causes\u00a0RBC\u00a0breakdown. Although there is no cure, most people with G6PD deficiency can lead healthy, normal lives by recognising triggers, avoiding high-risk medications and foods, and seeking timely medical care when symptoms occur.\u00a0<\/p>\n\n\n\n<h2 class=\"wp-block-heading\"><span class=\"ez-toc-section\" id=\"Frequently_Asked_Questions_FAQs\"><\/span><strong>Frequently Asked Questions (FAQs)<\/strong><span class=\"ez-toc-section-end\"><\/span><\/h2>\n\n\n\n<div class=\"schema-faq wp-block-yoast-faq-block\"><div class=\"schema-faq-section\" id=\"faq-question-1784896286208\"><strong class=\"schema-faq-question\"><strong>Is G6PD deficiency hereditary?<\/strong><\/strong> <p class=\"schema-faq-answer\">Yes,\u00a0G6PD deficiency is an inherited genetic condition caused by mutations in the G6PD gene. It is passed down in an X-linked pattern, which is why it is more common in males than females<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a><\/sup>.<\/p> <\/div> <div class=\"schema-faq-section\" id=\"faq-question-1784896311139\"><strong class=\"schema-faq-question\"><strong>Can G6PD go away with age?<\/strong>\u00a0<\/strong> <p class=\"schema-faq-answer\">No,\u00a0G6PD deficiency is a lifelong condition and does not go away with age<sup><a href=\"https:\/\/www.healthdirect.gov.au\/G6PD-deficiency\" target=\"_blank\" rel=\"noreferrer noopener\">9<\/a>,<a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/37753082\/\" target=\"_blank\" rel=\"noreferrer noopener\">14<\/a><\/sup>.\u00a0However, many people remain symptom-free throughout their lives if they avoid known triggers.\u00a0<\/p> <\/div> <div class=\"schema-faq-section\" id=\"faq-question-1784896327330\"><strong class=\"schema-faq-question\"><strong>What are the dangers of G6PD?<\/strong>\u00a0<\/strong> <p class=\"schema-faq-answer\">The main danger of G6PD deficiency is acute haemolytic anaemia, in which\u00a0RBCs break down rapidly after exposure to certain medications, infections, or foods such as fava beans. Severe cases can lead to jaundice, kidney injury, or, in newborns, serious complications such as kernicterus,\u00a0if left\u00a0unaddressed<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a>,<a href=\"https:\/\/www.healthdirect.gov.au\/G6PD-deficiency\" target=\"_blank\" rel=\"noreferrer noopener\">9<\/a><\/sup>.<\/p> <\/div> <div class=\"schema-faq-section\" id=\"faq-question-1784896362627\"><strong class=\"schema-faq-question\"><strong>What is the most common trigger of G6PD?<\/strong><\/strong> <p class=\"schema-faq-answer\">Infections are the most common trigger of haemolytic episodes in people with G6PD deficiency. Other common triggers include certain medications (such as some antimalarials), fava beans, and exposure to chemicals like naphthalene (mothballs)<sup><a href=\"https:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">1<\/a>,<a href=\"https:\/\/www.healthdirect.gov.au\/G6PD-deficiency\" target=\"_blank\" rel=\"noreferrer noopener\">9<\/a><\/sup>.\u00a0<\/p> <\/div> <\/div>\n\n\n\n<h2 class=\"wp-block-heading\"><span class=\"ez-toc-section\" id=\"References\"><\/span><strong>References<\/strong><span class=\"ez-toc-section-end\"><\/span><\/h2>\n\n\n\n<p>1. Mak GK, Shah M. Glucose-6-Phosphate Dehydrogenase Deficiency. In:\u00a0<em>StatPearls<\/em>. StatPearls Publishing; 2026. Accessed July 15, 2026. <a href=\"http:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/\" target=\"_blank\" rel=\"noreferrer noopener\">http:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/<\/a>\u00a0<\/p>\n\n\n\n<p>2. Bubp J, Jen M, Matuszewski K. Caring for Glucose-6-Phosphate Dehydrogenase (G6PD)\u2013Deficient Patients: Implications for Pharmacy.\u00a0<em>Pharm Ther<\/em>. 2015;40(9):572-574.\u00a0Accessed July 15, 2026.\u00a0<a href=\"https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC4571844\/\" target=\"_blank\" rel=\"noreferrer noopener\">https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC4571844\/<\/a>\u00a0<\/p>\n\n\n\n<p>3. Cappellini MD, Fiorelli G. Glucose-6-phosphate dehydrogenase deficiency.\u00a0<em>Lancet<\/em>. 2008;371(9606):64-74. doi:10.1016\/S0140-6736(08)60073-2\u00a0<a href=\"https:\/\/medlineplus.gov\/genetics\/condition\/glucose-6-phosphate-dehydrogenase-deficiency\/\" target=\"_blank\" rel=\"noreferrer noopener\">https:\/\/medlineplus.gov\/genetics\/condition\/glucose-6-phosphate-dehydrogenase-deficiency\/<\/a><\/p>\n\n\n\n<p>4. Koromina M, Pandi MT, van der Spek PJ, Patrinos GP, Lauschke VM. The ethnogeographic variability of genetic factors underlying G6PD deficiency.\u00a0<em>Pharmacol Res<\/em>. 2021;173:105904. doi:10.1016\/j.phrs.2021.105904\u00a0<a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/34551338\/\" target=\"_blank\" rel=\"noreferrer noopener\">https:\/\/pubmed.ncbi.nlm.nih.gov\/34551338\/<\/a><\/p>\n\n\n\n<p>5. Awab GR, Aaram F, Jamornthanyawat N, et al. Protective effect of Mediterranean-type glucose-6-phosphate dehydrogenase deficiency against Plasmodium vivax malaria.\u00a0<em>eLife<\/em>. 10:e62448. doi:10.7554\/eLife.62448\u00a0<a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/33543710\/\" target=\"_blank\" rel=\"noreferrer noopener\">https:\/\/pubmed.ncbi.nlm.nih.gov\/33543710\/<\/a><\/p>\n\n\n\n<p>6. Stanton RC. Glucose-6-Phosphate Dehydrogenase, NADPH, and Cell Survival.\u00a0<em>Iubmb Life<\/em>. 2012;64(5):362-369. doi:10.1002\/iub.1017\u00a0<a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/22431005\/\" target=\"_blank\" rel=\"noreferrer noopener\">https:\/\/pubmed.ncbi.nlm.nih.gov\/22431005\/<\/a><\/p>\n\n\n\n<p>7. Karafin MS, Francis RO. Impact of G6PD status on red cell storage and transfusion outcomes.\u00a0<em>Blood Transfus<\/em>. 2019;17(4):289-295. doi:10.2450\/2019.0092-19\u00a0 <a href=\"https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC6683872\/\" target=\"_blank\" rel=\"noreferrer noopener\">https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC6683872\/<\/a><\/p>\n\n\n\n<p>8. McDonagh EM, Thorn CF, Bautista JM, Youngster I, Altman RB, Klein TE. PharmGKB summary: very important pharmacogene information for G6PD.\u00a0<em>Pharmacogenet Genomics<\/em>. 2012;22(3):219-228. doi:10.1097\/FPC.0b013e32834eb313\u00a0<a href=\"https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC6683872\/\" target=\"_blank\" rel=\"noreferrer noopener\">https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC6683872\/<\/a><\/p>\n\n\n\n<p>9. Favism.\u00a0Healthdirect.\u00a0February 5, 2026. Accessed July 15, 2026. <a href=\"https:\/\/www.healthdirect.gov.au\/G6PD-deficiency\" target=\"_blank\" rel=\"noreferrer noopener\">https:\/\/www.healthdirect.gov.au\/G6PD-deficiency<\/a>\u00a0<\/p>\n\n\n\n<p>10. G6PD Test.\u00a0MedlinePlus Medical Test. Accessed July 15, 2026. <a href=\"https:\/\/medlineplus.gov\/lab-tests\/g6pd-test\/\" target=\"_blank\" rel=\"noreferrer noopener\">https:\/\/medlineplus.gov\/lab-tests\/g6pd-test\/<\/a>\u00a0<\/p>\n\n\n\n<p>11. Arese P, Gallo V, Pantaleo A, Turrini F. Life and Death of Glucose-6-Phosphate Dehydrogenase (G6PD) Deficient Erythrocytes \u2013 Role of Redox Stress and Band 3 Modifications.\u00a0<em>Transfus Med Hemotherapy<\/em>. 2012;39(5):328-334. doi:10.1159\/000343123\u00a0<a href=\"https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC3678266\/\" target=\"_blank\" rel=\"noreferrer noopener\">https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC3678266\/<\/a><\/p>\n\n\n\n<p>12. Garcia AA, Koperniku A, Ferreira JCB, Mochly-Rosen D. Treatment Strategies for Glucose-6-Phosphate Dehydrogenase Deficiency: Past and Future Perspectives.\u00a0<em>Trends Pharmacol Sci<\/em>. 2021;42(10):829-844. doi:10.1016\/j.tips.2021.07.002\u00a0<a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/34389161\/\" target=\"_blank\" rel=\"noreferrer noopener\">https:\/\/pubmed.ncbi.nlm.nih.gov\/34389161\/<\/a><\/p>\n\n\n\n<p>13. Ansong-Assoku B, Adnan M, Daley SF, Ankola PA. Neonatal Jaundice. In:\u00a0<em>StatPearls<\/em>. StatPearls Publishing; 2026. Accessed July 20, 2026. <a href=\"http:\/\/www.ncbi.nlm.nih.gov\/books\/NBK532930\/\" target=\"_blank\" rel=\"noreferrer noopener\">http:\/\/www.ncbi.nlm.nih.gov\/books\/NBK532930\/<\/a>\u00a0<\/p>\n\n\n\n<p>14. Israel A, Sch\u00e4ffer AA, Berkovitch M, et al. Glucose-6-Phosphate Dehydrogenase (G6PD) Deficiency and Long-Term Risk of Immune-Related diseases.\u00a0<em>medRxiv<\/em>. Published online March 24, 2023:2023.03.23.23287616. doi:10.1101\/2023.03.23.23287616\u00a0<a href=\"https:\/\/pubmed.ncbi.nlm.nih.gov\/37753082\/\" target=\"_blank\" rel=\"noreferrer noopener\">https:\/\/pubmed.ncbi.nlm.nih.gov\/37753082\/<\/a><\/p>\n\n\n\n<p><strong><em>Disclaimer<\/em><\/strong><em>: The information provided here is for educational\/awareness purposes only and is not intended to be a substitute for medical treatment by a healthcare professional and should not be relied upon to diagnose or treat any medical condition. The reader should consult a registered medical practitioner to determine the appropriateness of the information and before consuming any medication. PharmEasy does not provide any guarantee or warranty (express or implied) regarding the accuracy, adequacy, completeness, legality, reliability or usefulness of the information; and disclaims any liability arising thereof.<\/em><\/p>\n\n\n\n<p><em>Links and product recommendations in the information<\/em>\u00a0<em>provided here are advertisements of third-party products available on the website. PharmEasy does not make any representation on the accuracy or suitability of such products\/services. Advertisements do not influence the editorial decisions or content. The information in this blog is subject to change without notice. The authors and administrators reserve the right to modify, add, or remove content without notification. It is your responsibility to review this disclaimer regularly for any changes<\/em>.<\/p>\n<\/body><\/html>\n<div class=\"pld-like-dislike-wrap pld-custom\">\r\n    <div class=\"pld-like-wrap  pld-common-wrap\">\r\n    <a href=\"javascript:void(0)\" class=\"pld-like-trigger pld-like-dislike-trigger  \" title=\"Likes\" data-post-id=\"283967\" data-trigger-type=\"like\" data-restriction=\"cookie\" data-already-liked=\"0\">\r\n                            <img src=\"https:\/\/blog-images-1.pharmeasy.in\/blog\/production\/wp-content\/uploads\/2024\/08\/12132143\/like.png\" alt=\"Likes\" \/>\r\n            <\/a>\r\n    <span class=\"pld-like-count-wrap pld-count-wrap\">    <\/span>\r\n<\/div><div class=\"pld-dislike-wrap  pld-common-wrap\">\r\n    <a href=\"javascript:void(0)\" class=\"pld-dislike-trigger pld-like-dislike-trigger  \" title=\"Dislikes\" data-post-id=\"283967\" data-trigger-type=\"dislike\" data-restriction=\"cookie\" data-already-liked=\"0\">\r\n                            <img src=\"https:\/\/blog-images-1.pharmeasy.in\/blog\/production\/wp-content\/uploads\/2024\/08\/12132208\/dislike.png\" alt=\"Dislikes\" \/>\r\n            <\/a>\r\n    <span class=\"pld-dislike-count-wrap pld-count-wrap\"><\/span>\r\n<\/div><\/div>","protected":false},"excerpt":{"rendered":"Introduction Some inherited conditions can remain unnoticed for years, even when they have been present since birth.\u00a0Glucose-6-phosphate dehydrogenase (G6PD) deficiency is among the most common inherited enzyme disorders worldwide. Many people discover it only after certain medicines, foods, or infections trigger symptoms1,2.\u00a0 Most people\u00a0with G6PD deficiency live healthy, normal lives,\u00a0but\u00a0recognising triggers is\u00a0important for preventing\u00a0complications.\u00a0This\u00a0blog\u00a0covers\u00a0the symptoms, [\u2026]","protected":false},"author":176,"featured_media":284021,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"_acf_changed":false,"footnotes":"","_wp_rev_ctl_limit":""},"categories":[10435],"tags":[14578,14576,14577,14579],"acf":[],"_embedded":{"wp:featuredmedia":[{"source_url":"https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/G6PD-Deficiency.webp"}]},"_links":{"self":[{"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/posts\/283967"}],"collection":[{"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/posts"}],"about":[{"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/types\/post"}],"author":[{"embeddable":true,"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/users\/176"}],"replies":[{"embeddable":true,"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/comments?post=283967"}],"version-history":[{"count":8,"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/posts\/283967\/revisions"}],"predecessor-version":[{"id":284069,"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/posts\/283967\/revisions\/284069"}],"wp:featuredmedia":[{"embeddable":true,"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/media\/284021"}],"wp:attachment":[{"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/media?parent=283967"}],"wp:term":[{"taxonomy":"category","embeddable":true,"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/categories?post=283967"},{"taxonomy":"post_tag","embeddable":true,"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/tags?post=283967"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}