Did You Know?<\/p>\n\n\n\n

Around 5 million\u00a0old and damaged\u00a0RBCs\u00a0are cleared every second by macrophages\u00a0(immune cells).\u00a0These cells break down haemoglobin so its components can be recycled or removed from the body 1<\/a><\/sup>.\u00a0<\/li>\n\n\n\n
The iron released from old\u00a0RBCs\u00a0is recycled and used by the bone marrow to make new\u00a0RBCs 1<\/a><\/sup>.\u00a0<\/li>\n\n\n\n
Hemolysis\u00a0can occur inside blood vessels (intravascular\u00a0hemolysis) or in organs such as the spleen and liver (extravascular\u00a0hemolysis), depending on the underlying cause1<\/a>,3<\/a><\/sup>.\u00a0<\/li>\n<\/ul>\n<\/div><\/div>\n\n\n\n
<\/span>Causes of\u00a0Hemolysis<\/strong><\/span><\/h2>\n\n\n\n
Problems\u00a0can occur\u00a0when\u00a0RBCs\u00a0are destroyed faster than the bone marrow can replace them. This\u00a0usually\u00a0happens\u00a0for\u00a0two broad\u00a0reasons: intrinsic\u00a0causes, where the\u00a0issue is\u00a0within the\u00a0RBCs\u00a0themselves, and extrinsic\u00a0causes, where\u00a0something outside the\u00a0RBCs\u00a0damages them.\u00a0<\/p>\n\n\n\n
Intrinsic\u00a0(intracorpuscular)\u00a0Causes<\/strong><\/h3>\n\n\n\n
The defect is within the RBC itself, such as an abnormality in the RBC membrane, enzymes, or haemoglobin. While most intrinsic causes are inherited (present from birth), some may be\u00a0acquired\u00a0later in life.\u00a0<\/p>\n\n\n\n
\n
G6PD deficiency:<\/strong>\u00a0A genetic enzyme deficiency that makes\u00a0RBCs\u00a0vulnerable to certain medicines, infections, or foods such as fava beans1<\/a>,4<\/a><\/sup>.\u00a0<\/li>\n\n\n\n
Sickle cell disease:<\/strong>\u00a0An inherited disorder in which RBCs can become rigid and sickle-shaped under certain conditions, such as low oxygen levels, dehydration, or acidosis. These abnormal cells break down more easily than normal RBCs, leading to\u00a0hemolysis2<\/a><\/sup>.\u00a0<\/li>\n\n\n\n
Thalassaemia:<\/strong>\u00a0A group of inherited blood disorders that reduce haemoglobin production, leading to fragile\u00a0RBCs2<\/a><\/sup>.<\/li>\n\n\n\n
Hereditary spherocytosis:<\/strong>\u00a0A condition in which\u00a0RBCs\u00a0become sphere-shaped instead of their normal disc shape, making them more likely to be destroyed by the spleen1<\/a><\/sup>.\u00a0<\/li>\n\n\n\n
Pyruvate kinase deficiency:<\/strong>\u00a0A rare inherited enzyme disorder that\u00a0causes\u00a0RBCs\u00a0to break down faster than usual5<\/a><\/sup>.\u00a0<\/li>\n<\/ul>\n\n\n\n
Extrinsic\u00a0(extracorpuscular) Causes<\/strong><\/h3>\n\n\n\n
The RBCs are structurally normal, but they are destroyed by factors outside the RBC, such as immune reactions, infections, certain medications or toxins, mechanical injury, or an enlarged spleen. These causes usually develop later in life, although not all extrinsic causes are\u00a0acquired.\u00a0<\/p>\n\n\n\n
\n
Autoimmune haemolytic anaemia (AIHA):<\/strong>\u00a0The immune system mistakenly attacks and destroys healthy\u00a0RBCs6<\/a><\/sup>.\u00a0<\/li>\n\n\n\n
Infections:<\/strong>\u00a0Certain bacterial, viral, or parasitic infections, such as malaria, can trigger\u00a0hemolysis6<\/a>,7<\/a><\/sup>.\u00a0<\/li>\n\n\n\n
Blood transfusion reactions:\u00a0<\/strong>Receiving incompatible blood can cause rapid destruction of transfused\u00a0RBCs6<\/a><\/sup>.\u00a0<\/li>\n\n\n\n
Mechanical damage:<\/strong>\u00a0Artificial heart valves, ventricular assist devices, or prolonged strenuous exercise (such as marathon running) can physically damage\u00a0RBCs6<\/a>,8<\/a><\/sup>.\u00a0<\/li>\n\n\n\n
Microangiopathic haemolytic anaemia (MAHA):<\/strong>\u00a0RBCs\u00a0are damaged as they pass through narrowed or injured small blood vessels, as seen in conditions such as thrombotic thrombocytopenic purpura and haemolytic uraemic syndrome9<\/a><\/sup>.\u00a0<\/li>\n\n\n\n
Certain cancers:\u00a0<\/strong>Blood cancers such as lymphoma or chronic lymphocytic leukaemia may increase the risk of\u00a0hemolysis10<\/a><\/sup>.\u00a0<\/li>\n\n\n\n
Medications, toxins,\u00a0and chemicals:\u00a0<\/strong>Exposure to\u00a0certain medications, heavy metals, or certain industrial chemicals can damage\u00a0RBCs6<\/a><\/sup>.\u00a0<\/li>\n\n\n\n
Hypersplenism (hyperactive\u00a0spleen):<\/strong>\u00a0An enlarged spleen may\u00a0become hyperactive and\u00a0trap and destroy\u00a0RBCs\u00a0more rapidly than normal11<\/a><\/sup>.\u00a0<\/li>\n<\/ul>\n\n\n\n
Important:<\/strong>\u00a0Although intrinsic haemolytic anaemias are usually inherited and extrinsic haemolytic anaemias are often\u00a0acquired, this is not always the case. Some intrinsic causes may be\u00a0acquired, so the terms intrinsic and extrinsic do not strictly mean inherited and\u00a0acquired, respectively.\u00a0<\/p>\n\n\n\n
<\/span>Symptoms of\u00a0Hemolysis<\/strong><\/span><\/h2>\n\n\n\nSymptoms can vary from person to person. They may depend on how quickly\u00a0RBCs\u00a0are being destroyed, how severe the condition is, and what is causing it. Common\u00a0hemolysis\u00a0symptoms\u00a0and signs\u00a0include 6<\/a>,12<\/a><\/sup>:\u00a0<\/p>\n\n\n\n
\n
Fatigue or weakness\u00a0<\/li>\n\n\n\n
Pale skin (pallor)\u00a0<\/li>\n\n\n\n
Jaundice\u00a0(which causes yellowing of the skin\u00a0and\u00a0eyes)\u00a0<\/li>\n\n\n\n
Dark or reddish-brown urine\u00a0<\/li>\n\n\n\n
Shortness of breath\u00a0<\/li>\n\n\n\n
Rapid heartbeat (tachycardia)\u00a0<\/li>\n\n\n\n
Dizziness or light-headedness\u00a0<\/li>\n<\/ul>\n\n\n\n
Note:<\/strong>\u00a0Dark or reddish-brown urine is more commonly seen in intravascular\u00a0hemolysis\u00a0due to the presence of haemoglobin in the urine (haemoglobinuria). In extravascular\u00a0hemolysis, urine usually does not become dark because\u00a0RBCs\u00a0are broken down\u00a0mainly in\u00a0the spleen and liver. Dark urine\u00a0caused by haemoglobinuria should also be distinguished from haematuria (blood in the urine) and myoglobinuria (muscle protein in the urine).\u00a0<\/em><\/p>\n\n\n\n
<\/span>Types of\u00a0Hemolysis<\/strong>\u00a0<\/span><\/h2>\n\n\n\n
Doctors may describe\u00a0hemolysis\u00a0in\u00a0different ways, depending on where\u00a0RBCs\u00a0are destroyed and what is causing the destruction.\u00a0<\/p>\n\n\n\n
Based on the site of\u00a0RBC\u00a0destruction<\/strong><\/h3>\n\n\n\n\n
Extravascular\u00a0hemolysis:<\/strong>\u00a0RBCs\u00a0are\u00a0mainly removed\u00a0and broken down by immune cells (macrophages) in the spleen and liver. This is the most common type of\u00a0hemolysis.\u00a0<\/li>\n\n\n\n
Intravascular\u00a0hemolysis:<\/strong>\u00a0RBCs\u00a0are destroyed within the bloodstream1<\/a><\/sup>.\u00a0<\/li>\n<\/ul>\n\n\n\n
Based on the cause<\/strong><\/h3>\n\n\n\n\n
Inherited (intrinsic)\u00a0hemolysis:\u00a0<\/strong>Caused by genetic disorders that affect the\u00a0RBCs, such as G6PD deficiency, sickle cell disease, thalassaemia, and hereditary spherocytosis1<\/a>,2<\/a><\/sup>.<\/li>\n\n\n\n
Acquired (extrinsic)\u00a0hemolysis:<\/strong>\u00a0Caused by external factors, including autoimmune diseases, infections, certain medicines, blood transfusion reactions, toxins, or mechanical injury6<\/a><\/sup>.<\/li>\n<\/ul>\n\n\n\n
<\/span>How Is\u00a0Hemolysis\u00a0Diagnosed?<\/strong><\/span><\/h2>\n\n\n\nIf your doctor suspects\u00a0hemolysis, they may recommend a complete blood count<\/strong><\/a> to check for anaemia. They may also advise other blood or urine tests<\/strong><\/a>, such as6<\/a><\/sup>:\u00a0<\/p>\n\n\n\n
\n
Absolute reticulocyte count\u00a0<\/li>\n\n\n\n
Direct Coombs (antiglobulin) test<\/a>\u00a0\u00a0<\/strong><\/li>\n\n\n\n
Serum lactate dehydrogenase (LDH)<\/a>\u00a0<\/strong><\/li>\n\n\n\n
Serum haptoglobin level<\/strong><\/a>\u00a0<\/li>\n\n\n\n
Bilirubin test<\/strong>\u00a0<\/a><\/li>\n\n\n\n
Peripheral blood smear\u00a0<\/strong><\/a><\/li>\n<\/ul>\n\n\n\nAdditional tests, such as\u00a0 G6PD tests<\/strong><\/a>, may be recommended depending on the suspected cause.\u00a0<\/p>\n\n\n\n
<\/span>Treatment of\u00a0Hemolysis<\/strong><\/span><\/h2>\n\n\n\n
$\"Treatment$ <\/figure><\/div>\n\n\n
Treatment depends on the cause, how severe the\u00a0hemolysis\u00a0is, and the person\u2019s overall health. Your doctor may recommend one or more of the following options 4<\/a>,6<\/a>,13<\/a><\/sup>:\u00a0<\/p>\n\n\n\n
\n
Addressing\u00a0the underlying cause, such as an infection or autoimmune disease.\u00a0<\/li>\n\n\n\n
Stopping or changing the medicine causing\u00a0hemolysis, if applicable.\u00a0<\/li>\n\n\n\n
Blood transfusions to\u00a0manage\u00a0severe anaemia.\u00a0<\/li>\n\n\n\n
Supplements to support\u00a0RBC\u00a0production.\u00a0<\/li>\n\n\n\n
Supportive care, including intravenous fluids and oxygen therapy for severe cases.\u00a0<\/li>\n\n\n\n
Avoiding known triggers, such as certain medicines, infections, or fava beans, in people with G6PD deficiency.\u00a0<\/li>\n\n\n\n
Splenectomy (surgical removal of the spleen) in selected cases of chronic\u00a0hemolysis\u00a0(may increase the long-term risk of severe infections).\u00a0\u00a0<\/li>\n<\/ul>\n\n\n\n
<\/span>Possible Complications of\u00a0Hemolysis<\/strong><\/span><\/h2>\n\n\n\nIf\u00a0hemolysis\u00a0is severe or not treated, it may lead to complications such as 13<\/a>,14<\/a><\/sup>:\u00a0<\/p>\n\n\n\n
\n
Severe jaundice\u00a0<\/li>\n\n\n\n
Gallstones\u00a0<\/li>\n\n\n\n
Acute kidney injury\u00a0<\/li>\n\n\n\n
Heart complications due to severe or prolonged anaemia\u00a0<\/li>\n\n\n\n
Life-threatening haemolytic crisis in severe cases\u00a0<\/li>\n<\/ul>\n\n\n\n
<\/span>Prevention Tips\u00a0for\u00a0Hemolysis<\/strong><\/span><\/h2>\n\n\n\n $\"Prevention$ <\/figure>\n\n\n\n
Not every type of\u00a0hemolysis\u00a0can be prevented. However, the following steps may help lower the risk of severe episodes and related complications:\u00a0<\/p>\n\n\n\n
\n
Take medicines only as prescribed by your doctor.\u00a0<\/li>\n\n\n\n
Avoid known triggers, such as certain medicines, chemicals, or foods (e.g., fava beans in people with G6PD deficiency)4<\/a><\/sup>.<\/li>\n\n\n\n
Manage\u00a0infections promptly and keep vaccinations up to date.\u00a0<\/li>\n\n\n\n
Inform your doctor if you have a history of\u00a0hemolysis\u00a0or an inherited blood disorder.\u00a0<\/li>\n\n\n\n
Attend regular follow-up appointments and blood tests if you have a chronic haemolytic condition.\u00a0<\/li>\n\n\n\n
Seek medical attention early if you notice symptoms such as jaundice, dark urine, unusual fatigue, or shortness of breath.\u00a0<\/li>\n\n\n\n
Consider genetic counselling if you have a family history of inherited haemolytic disorders 13<\/a><\/sup>.<\/li>\n<\/ul>\n\n\n\n
<\/span>When Should You Consult a Doctor?<\/strong><\/span><\/h2>\n\n\n\nSpeak to a doctor<\/strong><\/a> if you notice any of the following symptoms, especially if they are new, persistent, or getting worse4<\/a>,6<\/a>,12<\/a>,13<\/a><\/sup>:<\/p>\n\n\n\n
\n
Persistent fatigue, weakness, or shortness of breath\u00a0<\/li>\n\n\n\n
Yellowing of the skin or eyes (jaundice)\u00a0<\/li>\n\n\n\n
Dark or reddish-brown urine\u00a0<\/li>\n\n\n\n
Pale skin or dizziness\u00a0<\/li>\n\n\n\n
Unexplained fever or chills\u00a0<\/li>\n\n\n\n
Pain or fullness in the upper left side of the abdomen (which may indicate an enlarged spleen)\u00a0<\/li>\n\n\n\n
Symptoms after starting a new medicine or following a blood transfusion\u00a0<\/li>\n\n\n\n
A family history of inherited blood disorders with new or worsening symptoms\u00a0<\/li>\n<\/ul>\n\n\n\n
Also Read: Folate Deficiency: Meaning, Causes, Symptoms, Treatment &\u00a0Prevention<\/a>\u00a0<\/em><\/strong><\/p>\n\n\n\n
<\/span>Conclusion<\/strong><\/span><\/h2>\n\n\n\n
Hemolysis\u00a0is a normal process when old\u00a0RBCs\u00a0are removed from the body. It becomes a concern when\u00a0RBCs\u00a0break down too early or too quickly. This may happen because of inherited blood disorders or acquired medical conditions. Mild cases may cause few or no symptoms, but severe cases can lead to serious complications. Recognising symptoms early, finding the cause, and getting timely treatment can make a meaningful difference. If you have symptoms that may suggest\u00a0hemolysis, speak to your doctor for the right diagnosis and care.\u00a0<\/p>\n\n\n\n
<\/span>Frequently Asked Questions (FAQs)<\/strong><\/span><\/h2>\n\n\n\nIs\u00a0hemolysis\u00a0a disease?<\/strong>\u00a0<\/strong> Hemolysis\u00a0simply means the breakdown of\u00a0RBCs, and it can be a normal body process. However, when\u00a0RBCs\u00a0break down too early or too quickly, it may point to an underlying medical condition that needs evaluation 1<\/a><\/sup>.<\/p> <\/div>
What can trigger\u00a0hemolysis?<\/strong>\u00a0<\/strong> Hemolysis\u00a0may be triggered by inherited blood disorders, autoimmune diseases, infections, certain medicines, blood transfusion reactions, toxins, mechanical heart valves, and, in people with G6PD deficiency, foods such as fava beans 4<\/a>,6<\/a><\/sup>.<\/p> <\/div>
How does\u00a0hemolysis\u00a0affect potassium levels?<\/strong><\/strong> When\u00a0RBCs\u00a0break down, they release potassium. Significant\u00a0hemolysis\u00a0can therefore increase blood potassium levels, although falsely elevated potassium (pseudohyperkalaemia) can also occur if blood cells break during sample collection 15<\/a>,16<\/a><\/sup>.\u00a0<\/p> <\/div>
Is\u00a0hemolysis\u00a0life-threatening?<\/strong>\u00a0<\/strong> Mild cases of premature or excessive\u00a0hemolysis\u00a0may not be serious, but severe cases could be life-threatening. They can lead to severe anaemia or other complications 1<\/a>,13<\/a><\/sup>.\u00a0This makes early medical treatment important.\u00a0<\/p> <\/div>
What foods cause\u00a0hemolysis?<\/strong><\/strong> Most foods do not cause\u00a0hemolysis. However, fava beans can trigger\u00a0hemolysis\u00a0in some people with G6PD deficiency. If you have this condition, ask your doctor which foods, medicines, or other triggers you should avoid 4<\/a><\/sup>.<\/p> <\/div>
Does\u00a0hemolysis\u00a0cause\u00a0hyperkalaemia?<\/strong>\u00a0<\/strong> Severe\u00a0hemolysis\u00a0may cause hyperkalaemia, which means high potassium levels in the blood, because potassium is released from damaged\u00a0RBCs. This needs medical evaluation, especially if symptoms such as muscle weakness or abnormal heart rhythms occur 15<\/a><\/sup>.<\/p> <\/div> <\/div>\n\n\n\n
<\/span>References<\/strong><\/span><\/h2>\n\n\n\n1. Thiagarajan P, Parker CJ, Prchal JT. How Do Red Blood Cells Die?\u00a0Front Physiol<\/em>. 2021;12:655393. doi:10.3389\/fphys.2021.655393\u00a0https:\/\/pubmed.ncbi.nlm.nih.gov\/33790808\/<\/a><\/p>\n\n\n\n
2. Zhong H, Yazdanbakhsh K. Hemolysis and immune regulation.\u00a0Curr Opin Hematol<\/em>. 2018;25(3):177-182. doi:10.1097\/MOH.0000000000000423\u00a0https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC6309361\/<\/a><\/p>\n\n\n\n
3. Hemolysis. MedlinePlus. Accessed July 20, 2026. https:\/\/medlineplus.gov\/ency\/article\/002372.htm<\/a>\u00a0<\/p>\n\n\n\n
4. Mak GK, Shah M. Glucose-6-Phosphate Dehydrogenase Deficiency.\u00a0StatPearls<\/em>. Accessed July 20, 2026. http:\/\/www.ncbi.nlm.nih.gov\/books\/NBK470315\/<\/a>\u00a0<\/p>\n\n\n\n
5. Higa S, Keapoletswe K, Cirneanu L, Hagenaars S, LI J, Zagadailov E. P1476: The Clinical Characteristics And Overall Survival Of Patients With Pyruvate Kinase Deficiency In The\u00a0UK: A Real-World Study.\u00a0HemaSphere<\/em>. 2023;7(Suppl):e0113950. doi:10.1097\/01.HS9.0000972788.01139.50\u00a0 https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC10430506\/<\/a><\/p>\n\n\n\n
6. Hemolytic anemia: MedlinePlus. Accessed July 20, 2026. https:\/\/medlineplus.gov\/ency\/article\/000571.htm\u00a0<\/a><\/p>\n\n\n\n
7. Thomas L.\u00a0Hemolysis\u00a0as Influence & Interference Factor.\u00a0EJIFCC<\/em>. 2002;13(4):95-98.\u00a0Accessed July 20, 2026. https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC6208064\/<\/a>\u00a0<\/p>\n\n\n\n
8. Patil HR, O\u2019Keefe JH, Lavie CJ, Magalski A, Vogel RA, McCullough PA. Cardiovascular Damage Resulting from Chronic Excessive Endurance Exercise.\u00a0Mo Med<\/em>. 2012;109(4):312-321.\u00a0Accessed July 20, 2026. https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC6179786\/<\/a>\u00a0<\/p>\n\n\n\n
9. Chaudhary P, Maharjan N, Subedi B. Microangiopathic Hemolytic Anemia as the Initial Presentation of Metastatic Signet-Ring Cell Carcinoma of the Colon: A Case Report.\u00a0Cureus<\/em>. 16(12):e76034. doi:10.7759\/cureus.76034\u00a0https:\/\/pubmed.ncbi.nlm.nih.gov\/39835047\/<\/a><\/p>\n\n\n\n
10. Tozluklu NN, G\u00fcven\u00e7 B. Autoimmune Hemolytic Anemia as the Presenting Feature of Chronic Lymphocytic Leukemia: Two Contrasting Cases Across Different Age Groups.\u00a0Hematol Transfus Cell Ther<\/em>. 2025;47:106115. doi:10.1016\/j.htct.2025.106115\u00a0https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC8616265\/<\/a><\/p>\n\n\n\n
11. Lv Y, Lau WY, Li Y, et al. Hypersplenism: History and current status.\u00a0Exp Ther Med<\/em>. 2016;12(4):2377-2382. doi:10.3892\/etm.2016.3683\u00a0https:\/\/pubmed.ncbi.nlm.nih.gov\/27703501\/<\/a><\/p>\n\n\n\n
12. Palmer D, Seviar D. How to approach\u00a0hemolysis: Haemolytic anaemia for the general physician.\u00a0Clin Med<\/em>. 2022;22(3):210-213. doi:10.7861\/clinmed.2022-0142\u00a0https:\/\/pubmed.ncbi.nlm.nih.gov\/35584830\/<\/a><\/p>\n\n\n\n
13. Turner J, Parsi M, Badireddy M. Anemia.\u00a0StatPearls<\/em>. Accessed July 20, 2026. http:\/\/www.ncbi.nlm.nih.gov\/books\/NBK499994\/<\/a>\u00a0<\/p>\n\n\n\n
14. Palmer D, Seviar D. How to approach\u00a0hemolysis: Haemolytic anaemia for the general physician.\u00a0Clin Med<\/em>. 2022;22(3):210-213. doi:10.7861\/clinmed.2022-0142\u00a0https:\/\/pubmed.ncbi.nlm.nih.gov\/35584830\/<\/a><\/p>\n\n\n\n
15. High potassium level.\u00a0MedlinePlus. Accessed July 20, 2026. https:\/\/medlineplus.gov\/ency\/article\/001179.htm<\/a>\u00a0<\/p>\n\n\n\n
16. Asirvatham JR, Moses V, Bjornson L. Errors in Potassium Measurement: A Laboratory Perspective for the Clinician.\u00a0North Am J Med Sci<\/em>. 2013;5(4):255-259. doi:10.4103\/1947-2714.110426\u00a0 https:\/\/pmc.ncbi.nlm.nih.gov\/articles\/PMC3662091\/<\/a><\/p>\n\n\n\n
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\r\n \r\n $\"Dislikes\"$ \r\n <\/a>\r\n <\/span>\r\n<\/div><\/div>","protected":false},"excerpt":{"rendered":"Introduction Did you know that your red blood cells\u00a0(RBCs)\u00a0usually live for about 120 days? At the end of their lifespan, they are naturally broken down and recycled by the body through a process called\u00a0hemolysis1.\u00a0 However, if\u00a0RBCs\u00a0are destroyed much earlier than expected, or in larger numbers than usual, it can cause problems. This may happen because [\u2026]","protected":false},"author":176,"featured_media":283964,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"_acf_changed":false,"footnotes":"[]","_wp_rev_ctl_limit":""},"categories":[10435],"tags":[14572,14570,14574,14573,14571],"acf":[],"_embedded":{"wp:featuredmedia":[{"source_url":"https:\/\/pharmeasy.in\/blog\/wp-content\/uploads\/2026\/07\/Hemolysis.webp"}]},"_links":{"self":[{"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/posts\/283929"}],"collection":[{"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/posts"}],"about":[{"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/types\/post"}],"author":[{"embeddable":true,"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/users\/176"}],"replies":[{"embeddable":true,"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/comments?post=283929"}],"version-history":[{"count":5,"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/posts\/283929\/revisions"}],"predecessor-version":[{"id":283963,"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/posts\/283929\/revisions\/283963"}],"wp:featuredmedia":[{"embeddable":true,"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/media\/283964"}],"wp:attachment":[{"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/media?parent=283929"}],"wp:term":[{"taxonomy":"category","embeddable":true,"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/categories?post=283929"},{"taxonomy":"post_tag","embeddable":true,"href":"https:\/\/pharmeasy.in\/blog\/wp-json\/wp\/v2\/tags?post=283929"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}